Institutionen för medicinsk biokemi och mikrobiologi

Institutionen för medicinsk ... Forskargrupper Cancer Andor Pivarcsi Publikationer

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Publikationer

A comprehensive analysis of coding and non-coding transcriptomic changes in cutaneous squamous cell carcinoma

Das Mahapatra, Kunal; Pasquali, Lorenzo; Søndergaard, Jonas Nørskov; Lapins, Jan et al.

Ingår i Scientific Reports, 2020.

DOI för A comprehensive analysis of coding and non-coding transcriptomic changes in cutaneous squamous cell carcinoma
A mannose-binding receptor is expressed on human keratinocytes and mediates killing of Candida albicans.

Szolnoky, G; Bata-Csörgö, Z; Kenderessy, A S; Kiss, M et al.

Ingår i Journal of Investigative Dermatology, s. 205-13, 2001.

DOI för A mannose-binding receptor is expressed on human keratinocytes and mediates killing of Candida albicans.
A novel mechanism for anti-EGFR antibody action involves chemokine-mediated leukocyte infiltration.

Hoffmann, Thomas K; Schirlau, Kerstin; Sonkoly, Enikö; Brandau, Sven et al.

Ingår i International Journal of Cancer, s. 2589-96, 2009.

DOI för A novel mechanism for anti-EGFR antibody action involves chemokine-mediated leukocyte infiltration.
Activation of toll-like receptors alters the microRNA expression profile of keratinocytes.

Meisgen, Florian; Xu Landén, Ning; Bouez, Charbel; Zuccolo, Michela et al.

Ingår i Experimental dermatology, s. 281-3, 2014.

DOI för Activation of toll-like receptors alters the microRNA expression profile of keratinocytes.
Advances in microRNAs: implications for immunity and inflammatory diseases.

Sonkoly, E; Pivarcsi, A

Ingår i Journal of Cellular and Molecular Medicine (Print), s. 24-38, 2009.

DOI för Advances in microRNAs: implications for immunity and inflammatory diseases.
Are BIC (miR-155) polymorphisms associated with eczema susceptibility?

Sääf, Annika; Kockum, Ingrid; Wahlgren, Carl-Fredrik; Xu, Ning et al.

Ingår i Acta Dermato-Venereologica, s. 366-7, 2013.

DOI för Are BIC (miR-155) polymorphisms associated with eczema susceptibility?
Budesonide, but not tacrolimus, affects the immune functions of normal human keratinocytes.

Kis, K; Bodai, L; Polyanka, H; Eder, K et al.

Ingår i International Immunopharmacology, s. 358-68, 2006.

DOI för Budesonide, but not tacrolimus, affects the immune functions of normal human keratinocytes.
CC chemokine ligand 18, an atopic dermatitis-associated and dendritic cell-derived chemokine, is regulated by staphylococcal products and allergen exposure.

Pivarcsi, Andor; Gombert, Michael; Dieu-Nosjean, Marie-Caroline; Lauerma, Antti et al.

Ingår i Journal of Immunology, s. 5810-7, 2004.

DOI för CC chemokine ligand 18, an atopic dermatitis-associated and dendritic cell-derived chemokine, is regulated by staphylococcal products and allergen exposure.
CCL1-CCR8 interactions: an axis mediating the recruitment of T cells and Langerhans-type dendritic cells to sites of atopic skin inflammation.

Gombert, Michael; Dieu-Nosjean, Marie-Caroline; Winterberg, Franziska; Bünemann, Erich et al.

Ingår i Journal of Immunology, s. 5082-91, 2005.

DOI för CCL1-CCR8 interactions: an axis mediating the recruitment of T cells and Langerhans-type dendritic cells to sites of atopic skin inflammation.
Changes in the level of serum microRNAs in patients with psoriasis after antitumour necrosis factor-α therapy.

Pivarcsi, A; Meisgen, F; Xu, N; Ståhle, M et al.

Ingår i British Journal of Dermatology, s. 563-70, 2013.

DOI för Changes in the level of serum microRNAs in patients with psoriasis after antitumour necrosis factor-α therapy.
Characterization of EGFR and ErbB2 expression in atopic dermatitis patients.

Sääf, Annika; Pivarcsi, Andor; Winge, Mårten C G; Wahlgren, Carl-Fredrik et al.

Ingår i Archives of Dermatological Research, s. 773-80, 2012.

DOI för Characterization of EGFR and ErbB2 expression in atopic dermatitis patients.
Chemokine networks in atopic dermatitis: traffic signals of disease.

Pivarcsi, Andor; Homey, Bernhard

Ingår i Current Allergy and Asthma Reports, s. 284-90, 2005.

DOI för Chemokine networks in atopic dermatitis: traffic signals of disease.
Chromatin interactions in differentiating keratinocytes reveal novel atopic dermatitis- and psoriasis-associated genes

Sahlén, Pelin; Spalinskas, Rapolas; Asad, Samina; Mahapatra, Kunal Das et al.

Ingår i Journal of Allergy and Clinical Immunology, s. 1742-1752, 2021.

DOI för Chromatin interactions in differentiating keratinocytes reveal novel atopic dermatitis- and psoriasis-associated genes Ladda ner fulltext (pdf) av Chromatin interactions in differentiating keratinocytes reveal novel atopic dermatitis- and psoriasis-associated genes

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Circulating microRNAs in extracellular vesicles as potential biomarkers for psoriatic arthritis in patients with psoriasis

Pasquali, L; Svedbom, A; Srivastava, A; Rosén, E et al.

Ingår i Journal of the European Academy of Dermatology and Venereology, s. 1248-1256, 2020.

DOI för Circulating microRNAs in extracellular vesicles as potential biomarkers for psoriatic arthritis in patients with psoriasis
Constraints for monocyte-derived dendritic cell functions under inflammatory conditions.

Fekete, Tünde; Szabo, Attila; Beltrame, Luca; Vivar, Nancy et al.

Ingår i European Journal of Immunology, s. 458-69, 2012.

DOI för Constraints for monocyte-derived dendritic cell functions under inflammatory conditions.
Cross-talk between IFN-γ and TWEAK through miR-149 amplifies skin inflammation in psoriasis

Srivastava, Ankit; Luo, Longlong; Lohcharoenkal, Warangkana; Meisgen, Florian et al.

Ingår i Journal of Allergy and Clinical Immunology, s. 2225-2235, 2021.

DOI för Cross-talk between IFN-γ and TWEAK through miR-149 amplifies skin inflammation in psoriasis Ladda ner fulltext (pdf) av Cross-talk between IFN-γ and TWEAK through miR-149 amplifies skin inflammation in psoriasis

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Differential expression of D-type cyclins in HaCaT keratinocytes and in psoriasis.

Belsõ, Nóra; Széll, Márta; Pivarcsi, Andor; Kis, Kornélia et al.

Ingår i Journal of Investigative Dermatology, s. 634-42, 2008.

DOI för Differential expression of D-type cyclins in HaCaT keratinocytes and in psoriasis.
Differentiation-regulated expression of Toll-like receptors 2 and 4 in HaCaT keratinocytes.

Pivarcsi, Andor; Koreck, Andrea; Bodai, Laszlo; Széll, Márta et al.

Ingår i Archives of Dermatological Research, s. 120-4, 2004.

DOI för Differentiation-regulated expression of Toll-like receptors 2 and 4 in HaCaT keratinocytes.
Distinct strains of Propionibacterium acnes induce selective human beta-defensin-2 and interleukin-8 expression in human keratinocytes through toll-like receptors.

Nagy, István; Pivarcsi, Andor; Koreck, Andrea; Széll, Márta et al.

Ingår i Journal of Investigative Dermatology, s. 931-8, 2005.

DOI för Distinct strains of Propionibacterium acnes induce selective human beta-defensin-2 and interleukin-8 expression in human keratinocytes through toll-like receptors.
Dithranol upregulates IL-10 receptors on the cultured human keratinocyte cell line HaCaT.

Farkas, A; Kemény, L; Szöny, B J; Bata-Csörgö, Z et al.

Ingår i Inflammation Research, s. 44-9, 2001.

DOI för Dithranol upregulates IL-10 receptors on the cultured human keratinocyte cell line HaCaT.
EGFR/Ras-induced CCL20 production modulates the tumour microenvironment.

Hippe, Andreas; Braun, Stephan Alexander; Oláh, Péter; Gerber, Peter Arne et al.

Ingår i British Journal of Cancer, s. 942-954, 2020.

DOI för EGFR/Ras-induced CCL20 production modulates the tumour microenvironment.
Expression and function of Toll-like receptors 2 and 4 in human keratinocytes.

Pivarcsi, Andor; Bodai, Laszlo; Réthi, Bence; Kenderessy-Szabó, Anna et al.

Ingår i International Immunology, s. 721-30, 2003.

DOI för Expression and function of Toll-like receptors 2 and 4 in human keratinocytes.
Extracellular microvesicle microRNAs as predictive biomarkers for targeted therapy in metastastic cutaneous malignant melanoma.

Svedman, Fernanda Costa; Lohcharoenkal, Warangkana; Bottai, Matteo; Brage, Suzanne Egyhazi et al.

Ingår i PLOS ONE, 2018.

DOI för Extracellular microvesicle microRNAs as predictive biomarkers for targeted therapy in metastastic cutaneous malignant melanoma.
Genetic polymorphisms altering microRNA activity in psoriasis--a key to solve the puzzle of missing heritability?

Pivarcsi, Andor; Ståhle, Mona; Sonkoly, Enikő

Ingår i Experimental dermatology, s. 620-4, 2014.

DOI för Genetic polymorphisms altering microRNA activity in psoriasis--a key to solve the puzzle of missing heritability?
Genome-Wide Screen for MicroRNAs Reveals a Role for miR-203 in Melanoma Metastasis.

Lohcharoenkal, Warangkana; Das Mahapatra, Kunal; Pasquali, Lorenzo; Crudden, Caitrin et al.

Ingår i Journal of Investigative Dermatology, s. 882-892, 2018.

DOI för Genome-Wide Screen for MicroRNAs Reveals a Role for miR-203 in Melanoma Metastasis.
Hemese, a hemocyte-specific transmembrane protein, affects the cellular immune response in Drosophila.

Kurucz, Eva; Zettervall, Carl-Johan; Sinka, Rita; Vilmos, Peter et al.

Ingår i Proceedings of the National Academy of Sciences of the United States of America, s. 2622-7, 2003.

DOI för Hemese, a hemocyte-specific transmembrane protein, affects the cellular immune response in Drosophila.
Histidine decarboxylase expression in human melanoma.

Haak-Frendscho, M; Darvas, Z; Hegyesi, H; Kárpáti, S et al.

Ingår i Journal of Investigative Dermatology, s. 345-52, 2000.

DOI för Histidine decarboxylase expression in human melanoma.
Human adult epidermal melanocytes cultured without chemical mitogens express the EGF receptor and respond to EGF.

Szabad, Gábor; Kormos, Bernadett; Pivarcsi, Andor; Széll, Márta et al.

Ingår i Archives of Dermatological Research, s. 191-200, 2007.

DOI för Human adult epidermal melanocytes cultured without chemical mitogens express the EGF receptor and respond to EGF.
Human antimicrobial protein hCAP18/LL-37 promotes a metastatic phenotype in breast cancer.

Weber, Günther; Chamorro, Clara Ibel; Granath, Fredrik; Liljegren, Annelie et al.

Ingår i Breast Cancer Research, 2009.

DOI för Human antimicrobial protein hCAP18/LL-37 promotes a metastatic phenotype in breast cancer.
Identification and characterization of a novel, psoriasis susceptibility-related noncoding RNA gene, PRINS.

Sonkoly, Eniko; Bata-Csorgo, Zsuzsanna; Pivarcsi, Andor; Polyanka, Hilda et al.

Ingår i Journal of Biological Chemistry, s. 24159-67, 2005.

DOI för Identification and characterization of a novel, psoriasis susceptibility-related noncoding RNA gene, PRINS.
Identification of chronological and photoageing-associated microRNAs in human skin.

Srivastava, Ankit; Karlsson, Magnus; Marionnet, Claire; Bernerd, Françoise et al.

Ingår i Scientific Reports, 2018.

DOI för Identification of chronological and photoageing-associated microRNAs in human skin.
Identification of novel non-coding RNA-based negative feedback regulating the expression of the oncogenic transcription factor GLI1.

Villegas, Victoria E; Rahman, Mohammed Ferdous-Ur; Fernandez-Barrena, Maite G; Diao, Yumei et al.

Ingår i Molecular Oncology, s. 912-26, 2014.

DOI för Identification of novel non-coding RNA-based negative feedback regulating the expression of the oncogenic transcription factor GLI1.
IL-31: a new link between T cells and pruritus in atopic skin inflammation.

Sonkoly, Eniko; Muller, Anja; Lauerma, Antti I; Pivarcsi, Andor et al.

Ingår i Journal of Allergy and Clinical Immunology, s. 411-7, 2006.

DOI för IL-31: a new link between T cells and pruritus in atopic skin inflammation.
Innate immune functions of the keratinocytes. A review.

Pivarcsi, A; Kemény, L; Dobozy, A

Ingår i Acta microbiologica et immunologica Hungarica, s. 303-10, 2004.

DOI för Innate immune functions of the keratinocytes. A review.
Interleukin-8 is regulated by miR-203 at the posttranscriptional level in primary human keratinocytes.

Wei, Tianling; Xu, Ning; Meisgen, Florian; Ståhle, Mona et al.

Ingår i EJD. European journal of dermatology, 2013.

DOI för Interleukin-8 is regulated by miR-203 at the posttranscriptional level in primary human keratinocytes.
Microbial compounds induce the expression of pro-inflammatory cytokines, chemokines and human beta-defensin-2 in vaginal epithelial cells.

Pivarcsi, Andor; Nagy, Istvan; Koreck, Andrea; Kis, Kornelia et al.

Ingår i Microbes and infection, s. 1117-27, 2005.

DOI för Microbial compounds induce the expression of pro-inflammatory cytokines, chemokines and human beta-defensin-2 in vaginal epithelial cells.
MicroRNA-125b down-regulates matrix metallopeptidase 13 and inhibits cutaneous squamous cell carcinoma cell proliferation, migration, and invasion.

Xu, Ning; Zhang, Lingyun; Meisgen, Florian; Harada, Masako et al.

Ingår i Journal of Biological Chemistry, s. 29899-908, 2012.

DOI för MicroRNA-125b down-regulates matrix metallopeptidase 13 and inhibits cutaneous squamous cell carcinoma cell proliferation, migration, and invasion.
MicroRNA-132 enhances transition from inflammation to proliferation during wound healing.

Li, Dongqing; Wang, Aoxue; Liu, Xi; Meisgen, Florian et al.

Ingår i Journal of Clinical Investigation, s. 3008-26, 2015.

DOI för MicroRNA-132 enhances transition from inflammation to proliferation during wound healing.
MicroRNA-132 promotes fibroblast migration via regulating RAS p21 protein activator 1 in skin wound healing.

Li, Xi; Li, Dongqing; Wikstrom, Jakob D; Pivarcsi, Andor et al.

Ingår i Scientific Reports, 2017.

DOI för MicroRNA-132 promotes fibroblast migration via regulating RAS p21 protein activator 1 in skin wound healing.
MicroRNA-132 with Therapeutic Potential in Chronic Wounds.

Li, Xi; Li, Dongqing; Wang, Aoxue; Chu, Tongbin et al.

Ingår i Journal of Investigative Dermatology, s. 2630-2638, 2017.

DOI för MicroRNA-132 with Therapeutic Potential in Chronic Wounds.
MicroRNA-146a suppresses IL-17-mediated skin inflammation and is genetically associated with psoriasis.

Srivastava, Ankit; Nikamo, Pernilla; Lohcharoenkal, Warangkana; Li, Dongqing et al.

Ingår i Journal of Allergy and Clinical Immunology, s. 550-561, 2017.

DOI för MicroRNA-146a suppresses IL-17-mediated skin inflammation and is genetically associated with psoriasis.
MicroRNA-203 functions as a tumor suppressor in basal cell carcinoma.

Sonkoly, E; Lovén, J; Xu, N; Meisgen, F et al.

Ingår i Oncogenesis, 2012.

DOI för MicroRNA-203 functions as a tumor suppressor in basal cell carcinoma.
MicroRNA-203 Inversely Correlates with Differentiation Grade, Targets c-MYC, and Functions as a Tumor Suppressor in cSCC.

Lohcharoenkal, Warangkana; Harada, Masako; Lovén, Jakob; Meisgen, Florian et al.

Ingår i Journal of Investigative Dermatology, s. 2485-2494, 2016.

DOI för MicroRNA-203 Inversely Correlates with Differentiation Grade, Targets c-MYC, and Functions as a Tumor Suppressor in cSCC.
MicroRNA-31 is overexpressed in cutaneous squamous cell carcinoma and regulates cell motility and colony formation ability of tumor cells.

Wang, Aoxue; Landén, Ning Xu; Meisgen, Florian; Lohcharoenkal, Warangkana et al.

Ingår i PLOS ONE, 2014.

DOI för MicroRNA-31 is overexpressed in cutaneous squamous cell carcinoma and regulates cell motility and colony formation ability of tumor cells.
MicroRNA-31 is overexpressed in psoriasis and modulates inflammatory cytokine and chemokine production in keratinocytes via targeting serine/threonine kinase 40.

Xu, Ning; Meisgen, Florian; Butler, Lynn M; Han, Gangwen et al.

Ingår i Journal of Immunology, s. 678-88, 2013.

DOI för MicroRNA-31 is overexpressed in psoriasis and modulates inflammatory cytokine and chemokine production in keratinocytes via targeting serine/threonine kinase 40.
MicroRNA-31 Promotes Skin Wound Healing by Enhancing Keratinocyte Proliferation and Migration.

Li, Dongqing; Li, X I; Wang, Aoxue; Meisgen, Florian et al.

Ingår i Journal of Investigative Dermatology, s. 1676-1685, 2015.

DOI för MicroRNA-31 Promotes Skin Wound Healing by Enhancing Keratinocyte Proliferation and Migration.
MicroRNAs and immunity: novel players in the regulation of normal immune function and inflammation.

Sonkoly, Enikö; Ståhle, Mona; Pivarcsi, Andor

Ingår i Seminars in Cancer Biology, s. 131-40, 2008.

DOI för MicroRNAs and immunity: novel players in the regulation of normal immune function and inflammation.
microRNAs in inflammation.

Sonkoly, Enikö; Pivarcsi, Andor

Ingår i International Reviews of Immunology, s. 535-61, 2009.

DOI för microRNAs in inflammation.
MicroRNAs in inflammation and response to injuries induced by environmental pollution.

Sonkoly, Enikö; Pivarcsi, Andor

Ingår i Mutation research, s. 46-53, 2011.

DOI för MicroRNAs in inflammation and response to injuries induced by environmental pollution.
MicroRNAs: novel regulators in skin inflammation.

Sonkoly, E; Ståhle, M; Pivarcsi, A

Ingår i Clincal and Experimental Dermatology, s. 312-5, 2008.

DOI för MicroRNAs: novel regulators in skin inflammation.
MicroRNAs: novel regulators involved in the pathogenesis of psoriasis?

Sonkoly, Enikö; Wei, Tianling; Janson, Peter C J; Sääf, Annika et al.

Ingår i PLOS ONE, 2007.

DOI för MicroRNAs: novel regulators involved in the pathogenesis of psoriasis?
miR ‐378a regulates keratinocyte responsiveness to interleukin‐17A in psoriasis

Xia, Ping; Pasquali, Lorenzo; Gao, Chenying; Srivastava, Ankit et al.

Ingår i British Journal of Dermatology, s. 211-222, 2022.

DOI för miR ‐378a regulates keratinocyte responsiveness to interleukin‐17A in psoriasis Ladda ner fulltext (pdf) av miR ‐378a regulates keratinocyte responsiveness to interleukin‐17A in psoriasis

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MiR-125b, a microRNA downregulated in psoriasis, modulates keratinocyte proliferation by targeting FGFR2.

Xu, Ning; Brodin, Petter; Wei, Tianling; Meisgen, Florian et al.

Ingår i Journal of Investigative Dermatology, s. 1521-9, 2011.

DOI för MiR-125b, a microRNA downregulated in psoriasis, modulates keratinocyte proliferation by targeting FGFR2.
MiR-130a Acts as a Tumor Suppressor MicroRNA in Cutaneous Squamous Cell Carcinoma and Regulates the Activity of the BMP/SMAD Pathway by Suppressing ACVR1

Lohcharoenkal, Warangkana; Li, Chen; Mahapatra, Kunal Das; Lapins, Jan et al.

Ingår i Journal of Investigative Dermatology, s. 1922-1931, 2021.

DOI för MiR-130a Acts as a Tumor Suppressor MicroRNA in Cutaneous Squamous Cell Carcinoma and Regulates the Activity of the BMP/SMAD Pathway by Suppressing ACVR1 Ladda ner fulltext (pdf) av MiR-130a Acts as a Tumor Suppressor MicroRNA in Cutaneous Squamous Cell Carcinoma and Regulates the Activity of the BMP/SMAD Pathway by Suppressing ACVR1

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MiR-146a negatively regulates TLR2-induced inflammatory responses in keratinocytes.

Meisgen, Florian; Xu Landén, Ning; Wang, Aoxue; Réthi, Bence et al.

Ingår i Journal of Investigative Dermatology, s. 1931-1940, 2014.

DOI för MiR-146a negatively regulates TLR2-induced inflammatory responses in keratinocytes.
MiR-155 is overexpressed in patients with atopic dermatitis and modulates T-cell proliferative responses by targeting cytotoxic T lymphocyte-associated antigen 4.

Sonkoly, Enikö; Janson, Peter; Majuri, Marja-Leena; Savinko, Terhi et al.

Ingår i Journal of Allergy and Clinical Immunology, s. 581-9.e1, 2010.

DOI för MiR-155 is overexpressed in patients with atopic dermatitis and modulates T-cell proliferative responses by targeting cytotoxic T lymphocyte-associated antigen 4.
miR-193b/365a cluster controls progression of epidermal squamous cell carcinoma.

Gastaldi, Cécile; Bertero, Thomas; Xu, Ning; Bourget-Ponzio, Isabelle et al.

Ingår i Carcinogenesis, s. 1110-20, 2014.

DOI för miR-193b/365a cluster controls progression of epidermal squamous cell carcinoma.
miR-19a/b and miR-20a promote wound healing by regulating the inflammatory response of keratinocytes

Li, Dongqing; Peng, Hongmei; Qu, Le; Sommar, Pehr et al.

Ingår i Journal of Investigative Dermatology, s. 659-671, 2021.

DOI för miR-19a/b and miR-20a promote wound healing by regulating the inflammatory response of keratinocytes Ladda ner fulltext (pdf) av miR-19a/b and miR-20a promote wound healing by regulating the inflammatory response of keratinocytes

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MiR-21 is up-regulated in psoriasis and suppresses T cell apoptosis.

Meisgen, Florian; Xu, Ning; Wei, Tianling; Janson, Peter C et al.

Ingår i Experimental dermatology, s. 312-4, 2012.

DOI för MiR-21 is up-regulated in psoriasis and suppresses T cell apoptosis.
MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma.

Lovén, Jakob; Zinin, Nikolay; Wahlström, Therese; Müller, Inga et al.

Ingår i Proceedings of the National Academy of Sciences of the United States of America, s. 1553-8, 2010.

DOI för MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma.
Negative regulatory effect of histamine in DNFB-induced contact hypersensitivity.

Garaczi, Edina; Széll, Márta; Jánossy, Tamás; Koreck, Andrea et al.

Ingår i International Immunology, s. 1781-8, 2004.

DOI för Negative regulatory effect of histamine in DNFB-induced contact hypersensitivity.
Next-generation sequencing identifies microRNAs that associate with pathogenic autoimmune neuroinflammation in rats.

Bergman, Petra; James, Tojo; Kular, Lara; Ruhrmann, Sabrina et al.

Ingår i Journal of Immunology, s. 4066-75, 2013.

DOI för Next-generation sequencing identifies microRNAs that associate with pathogenic autoimmune neuroinflammation in rats.
Next-Generation Sequencing Identifies the Keratinocyte-Specific miRNA Signature of Psoriasis.

Srivastava, Ankit; Meisgen, Florian; Pasquali, Lorenzo; Munkhammar, Sara et al.

Ingår i Journal of Investigative Dermatology, s. 2547-2550.e12, 2019.

DOI för Next-Generation Sequencing Identifies the Keratinocyte-Specific miRNA Signature of Psoriasis.
Proliferating keratinocytes are putative sources of the psoriasis susceptibility-related EDA+ (extra domain A of fibronectin) oncofetal fibronectin.

Széll, Márta; Bata-Csörgo, Zsuzsanna; Koreck, Andrea; Pivarcsi, Andor et al.

Ingår i Journal of Investigative Dermatology, s. 537-46, 2004.

DOI för Proliferating keratinocytes are putative sources of the psoriasis susceptibility-related EDA+ (extra domain A of fibronectin) oncofetal fibronectin.
Propionibacterium acnes and lipopolysaccharide induce the expression of antimicrobial peptides and proinflammatory cytokines/chemokines in human sebocytes.

Nagy, István; Pivarcsi, Andor; Kis, Kornélia; Koreck, Andrea et al.

Ingår i Microbes and infection, s. 2195-205, 2006.

DOI för Propionibacterium acnes and lipopolysaccharide induce the expression of antimicrobial peptides and proinflammatory cytokines/chemokines in human sebocytes.
Protein kinase C-dependent upregulation of miR-203 induces the differentiation of human keratinocytes

Sonkoly, Enikö; Wei, Tianling; Pavez Loriè, Elizabeth; Suzuki, Hiroyuki et al.

Ingår i Journal of Investigative Dermatology, s. 124-134, 2010.

DOI för Protein kinase C-dependent upregulation of miR-203 induces the differentiation of human keratinocytes
RNA editing of the GLI1 transcription factor modulates the output of Hedgehog signaling.

Shimokawa, Takashi; Rahman, Mohammed Ferdous-Ur; Tostar, Ulrica; Sonkoly, Enikö et al.

Ingår i RNA Biology, s. 321-33, 2013.

DOI för RNA editing of the GLI1 transcription factor modulates the output of Hedgehog signaling.
Serum factors regulate the expression of the proliferation-related genes alpha5 integrin and keratin 1, but not keratin 10, in HaCaT keratinocytes.

Pivarcsi, A; Széll, M; Kemény, L; Dobozy, A et al.

Ingår i Archives of Dermatological Research, s. 206-13, 2001.

DOI för Serum factors regulate the expression of the proliferation-related genes alpha5 integrin and keratin 1, but not keratin 10, in HaCaT keratinocytes.
The expression of keratinocyte growth factor receptor (FGFR2-IIIb) correlates with the high proliferative rate of HaCaT keratinocytes.

Nagy, Nikoletta; Bata-Csörgo, Zsuzsanna; Kopasz, Norbert; Szeg, Csilla et al.

Ingår i Experimental dermatology, s. 596-605, 2006.

DOI för The expression of keratinocyte growth factor receptor (FGFR2-IIIb) correlates with the high proliferative rate of HaCaT keratinocytes.
The expression of microRNA-203 during human skin morphogenesis.

Wei, Tianling; Orfanidis, Kyriakos; Xu, Ning; Janson, Peter et al.

Ingår i Experimental dermatology, s. 854-6, 2010.

DOI för The expression of microRNA-203 during human skin morphogenesis.
The human antimicrobial peptide LL-37 suppresses apoptosis in keratinocytes.

Chamorro, Clara I; Weber, Günther; Grönberg, Alvar; Pivarcsi, Andor et al.

Ingår i Journal of Investigative Dermatology, s. 937-44, 2009.

DOI för The human antimicrobial peptide LL-37 suppresses apoptosis in keratinocytes.
The Keratinocyte Transcriptome in Psoriasis: Pathways Related to Immune Responses, Cell Cycle and Keratinization.

Pasquali, Lorenzo; Srivastava, Ankit; Meisgen, Florian; Das Mahapatra, Kunal et al.

Ingår i Acta Dermato-Venereologica, s. 196-205, 2019.

DOI för The Keratinocyte Transcriptome in Psoriasis: Pathways Related to Immune Responses, Cell Cycle and Keratinization.
The role of innate immunity in the pathogenesis of acne.

Koreck, A; Pivarcsi, A; Dobozy, A; Kemény, L

Ingår i Dermatology, s. 96-105, 2003.

DOI för The role of innate immunity in the pathogenesis of acne.
Tofacitinib Represses the Janus Kinase-Signal Transducer and Activators of Transcription Signalling Pathway in Keratinocytes.

Srivastava, Ankit; Ståhle, Mona; Pivarcsi, Andor; Sonkoly, Enikö

Ingår i Acta Dermato-Venereologica, s. 772-775, 2018.

DOI för Tofacitinib Represses the Janus Kinase-Signal Transducer and Activators of Transcription Signalling Pathway in Keratinocytes.
Toll-like receptor 9-independent suppression of skin inflammation by oligonucleotides.

Pivarcsi, Andor

Ingår i Journal of Investigative Dermatology, s. 746-8, 2007.

DOI för Toll-like receptor 9-independent suppression of skin inflammation by oligonucleotides.
Tumor immune escape by the loss of homeostatic chemokine expression.

Pivarcsi, Andor; Müller, Anja; Hippe, Andreas; Rieker, Juliane et al.

Ingår i Proceedings of the National Academy of Sciences of the United States of America, s. 19055-60, 2007.

DOI för Tumor immune escape by the loss of homeostatic chemokine expression.